(4) Herein, we present a patient with a small meningioma concomitant with a disproportionately large and extensive hemispheric
brain edema. The patient had a good prognosis after surgical resection of the mass.
Brain computed tomography showed massive
brain edema. Empirical antibiotic treatment was continued with piperacillin/tazobactam (PTZ) 4.5 g every eight hours and clarithromycin 500 mg every 12 hours, and the
brain edema was treated with mannitol and dexamethasone.
For this reason, the enlargement of ONSD is seen as an early and first indicator of
brain edema, before the development of papillary stasis (2).
Peritumoral
brain edema (PTBE) is formed in 40%-78% of all cases of intracranial meningiomas.
The formation of
brain edema (BE), a continuous process with an intense intracellular and extracellular ion and water exchange [1], is one of the most important acute/subacute complications of cerebral injuries.
After bTBI,
brain edema reached the highest peak at day 3 (82.5% [+ or -] 2.1% vs.
The effect of hematoma removal for reducing the development of
brain edema in cases of putaminal hemorrhage.
It describes an unusual case of invasive aspergillosis of the paranasal sinuses involving the brain in a sickle cell disease (SCD) patient who eventually died because of post-craniotomy Salmonella ventriculitis, abscess collections and severe
brain edema. The rapid clinical progression and complications of invasive sinusitis presented in this case highlight the importance of early diagnosis and initiation of antifungal treatment in such cases.
Brain edema is the most common central nervous system (CNS) complication which occurs in approximately 0.5-1% of cases with diabetic ketoacidosis (DKA) and has a 20% mortality rate (1,2).
After doctors examined the baby at the hospital, they found that she had suffered a skull fracture and symptoms of
brain edema. When questioned by police, Kansai said that a month ago when he was not paying attention, his daughter "had fallen on her head, but fortunately she was not badly hurt," reported ETtoday.
(10) The causes of SBI are attributed to decreases in brain oxygen supply due to hypotension, hypoxia,
brain edema, intracranial hypertension, or anomalies in brain bloodstream; (2) it is defined as the response to a primary injury, brain cellular changes, brain ischemia, or
brain edema that is causing damage to neighboring brain tissue.
Moreover, the brain tissue was severely damaged during the operation, and the
brain edema was aggravated and the infection rate increased after operation.